School
Failure.-
Evaluation
and Treatment
Elena Boder, M.D.
From the Departments of Pediatrics, University of California at Los Angeles and Cedars-Sinai Medical Center, and the Division of Neurology, Childrens Hospital of Los Angeles. Los Angeles, California
Children who are failing in school have multiple problems and are clearly a
heteroge-neous group, both etiobogically and clinically.
They have a wide variety of neurodevelopmental deviations-in behavior, motor function,
percep-lion, language, and learning-which has led to the
concept of maturational lag, commonly referred to as “minimal brain dysfunction” (MBD).
Nevertheless, these children present initially with
problems either in behavior or scholastic
under-achievement, or-by far the largest group-as a
combination of both. The most common
behav-ioral problem leading to school failure in the early
grades is the hyperkinetic syndrome, inasmuch as
this behavioral manifestation is one of the least
likely to be tolerated in the school. In my
experi-ence, the most common and least recognized
cause of chronic underachievement at all grade
levels is specific learning disability, notably
devel-opmental
The purpose of this paper is to outline an
overall neuropsychoeducational approach to the
diagnosis and management of children referred
for school failure that I have found useful in a
private neuropediatric consultation practice.
THE OVERALL DIAGNOSTIC APPROACH
The essentials of the neuropsychoeducational
approach, in which the interdependence of
neurological, psychological, and educational
fac-tors is accepted as fundamental, are: orderly,
comprehensive diagnostic evaluation, utilizing a
multidisciplinary team of consultants as a basis for coordinated total management; close
communi-cation with key school personnel for diagnostic
interpretation and special educational planning;
parent and child counseling for diagnostic and
therapeutic interpretation, emphasizing the need
for a structured home routine; and psychotropic
drug therapy and psychotherapy as indicated.
If the child presents with a behavioral problem,
with or without concomitant scholastic
under-achievement, the initial diagnostic objective is to
determine whether the behavioral problem is
primarily neurological (neurodevelopmental),
primarily psychiatric, or both neurogenic and
psychogenic.
The next objective is to determine whether the
learning problem underlying the
underachieve-ment is a primary, specific learning disability or is
nonspecific and secondary to behavioral, mental,
emotional, motivational, sociocultural, and other
factors.
A third objective in the comprehensive
evalua-tion is to rule out all physical, neurological, and
peripheral sensory factors that may be causative
or contributory in the child’s school problems.
Most commonly, the presenting complaint of a
child referred for school failure is a behavioral
problem. The child’s underachievement, when
mentioned, is usually regarded as secondary to the
behavioral problem or to poor motivation.
Although it is generally recognized that the
#{176}Theterms dyslexia, specific dyslexia, and specific develop-mental dyslexia are used here interchangeably with
develop-mental dyslexia and specific, or primary, reading disability.
None is used as a broad term encompassing nonspecific reading disorders.
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Beverly Hills, California 90210.
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395hyperkinetic syndrome is frequently associated
with
secondary, nonspecific learning disabilities,it
has
not been sufficiently recognized that it isalso frequently associated with primary or
specific learning disabilities, notably
develop-mental dyslexia. Systematic screening for
devel-opmental
dyslexia is therefore an integral part ofmy expanded neurological examination of every
child referred for school failure, especially a child
with the hyperkmnetic syndrome.
It is taken for granted that the child failing in
school will invariably have a secondary, reactive
emotional overlay, and an attempt is made to
assess its severity. It is also important to ascertain
whether a primary emotional problem exists as a
causal or contributory factor in the child’s
under-achievement. However, the presence of an
emotional problem, whether primary or
second-ary or both, does not exclude the possibility that
the
child may also have developmental dyslexia.Therefore, sorting out the primary and secondary
emotional factors, though essential in developing
a therapeutic program for the child’s emotional
problems, is not essential in making a diagnosis of
developmental dyslexia or any other specific
learning disability.
OFFICE PROCEDURES
Children
referred for neuropediatric evaluationof school failure often have had a number of
previous diagnostic work-ups. A preliminary step
therefore is to obtain through the parents,
prefer-ably before the child’s first office visit, reports on
previous medical and psychological evaluations,
as well as past and present school reports,
includ-fig reports from the child’s current teacher on his
classroom behavior, scholastic achievement, and peer group interaction.
My evaluation ordinarily requires two or at
most three extended office visits, at which both
parents are asked to be present. Their presence at
the first visit is important not only to obtain a
more complete history and to observe family
interactions, but also to explore what each
parent’s main concerns about the child are, what
each believes to be the causes of the child’s
problems, and what they hope to gain from the
present consultation. The special advantage of
both
parents being present at the second visit isthat each can have a first-hand interpretation of
the
diagnosis and the proposed plan ofmanage-ment, which helps to ensure optimum
coopera-tion.
The first office visit begins with a brief
inter-view with the child in the presence of his parents,
based on a few simple questions about his
reac-tions to school. This provides an opportunity to
observe his general pattern of behavior and
reveals whether he recognizes his school
problems or denies them. Next, while the history
is being obtained from the parents, the child goes
to another room where the office assistant
admin-isters a set of paper-pencil tests and my diagnostic
screening procedure for developmental dyslexia,
which is outlined later.
The review of past records is supplemented by
a detailed medical, developmental, and family
history, giving emphasis to events which are
potential causative factors of brain damage,
noting any significant delays in speech and
language
and
motor skills, and inquiring aboutdyslexia and speech or language impairments in
siblings and other members of the family. It is
often difficult to elicit a positive history of
dyslexia in the parents; they will admit much
more readily to being poor spellers than slow
readers, probably because the irregularity of
English orthography makes poor spelling socially
acceptable.
Unless the child’s school problems are
rela-tively uncomplicated, the physical and
neurolog-ical examination usually is scheduled for the
second visit. It has been found advantageous to
have the parents present during the examination.
Their presence, which is reassuring to a younger
child, gives the examiner a chance to point out
the child’s assets and to let the parents see for
themselves some of the deficits in performance.
Later interpretation of the neurological findings
becomes more meaningful when it is related to
the parents’ own observations.
A
complete
physical examination is consideredan essential preliminary step to the expanded
neurological examination. Especially important
to rule out are physical defects, including gross
visual and auditory impairments, congenital
anomalies, abnormal somatic growth and
devel-opment,
allergic or endocrine disorders, chronicillness, and malnutrition.
Because of the variety of “soft” signs pointing
to maturational lags in children with school
difficulties, each child is given an expanded
neurological examination. Space does not permit
discussion of the growing number and variety of
special neurological tests designed to elicit soft
signs. The reader is referred to the other
discus-sions in this section. In expanding the routine
neurological examination, I limit myself as
required by the time available to tests I have
found especially valuable in eliciting precisely
those signs, soft or hard, which are directly
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relevant to school performance or point to the
other neurodevelopmental disorders frequently
associated with the hyperkinetic syndrome or
with developmental dyslexia.
Among the tests in my expanded neurological
examination are standard tests of fine and gross
motor coordination and manual praxis, including
dressing and undressing; tests for cerebellar
func-tion, lateral dominance, including Schilder’s
arm-extension test, finger gnosia, and stereognosis;
Benton
right-left discrimination test;color-naming test; reciting the days of the week as a test
for auditory sequential memory;
sentence-repeti-tion test for articulation and short-term memory;
and the paper-pencil tests administered at the
first office visit along with my diagnostic
screen-ing procedure for development dyslexia. The last
is repeated at the second visit to check the
consistency of the results.
The paper-pencil tests include copying of
geometric designs to point up visual-perceptual
and visuomotor coordination impairments;
draw-ing “a house, a man, and a tree,” a picture of the
child’s own family, the face of a clock from
memory, and spontaneous drawings, which may
disclose immature performance, bizarre
dispro-portions, distorted body-image, and visual-spatial
deficits. The family drawing may also provide
valuable clues to family interactions and the
child’s self-image.
The child’s general pattern of behavior has also
been noted by the end of the neurological
exami-nation, along with the presence of involuntary
movements, such as choreiform movements,
tremors, or tics, and the stereotyped movements
typical of the schizophrenic child.
Diagnostic Screening Test for Developmental Dyslexia
An integral part of the expanded neurological
examination is my empirically evolved Diagnostic
Screening Test for Developmental Dyslexia.
Described in detail elsewhere,’-2 it provides an
approach to differentiate specific dyslexia from
nonspecific reading retardation. Such
differentia-tion has immediate practical bearing since
specific dyslexia calls for remedial reading
tech-niques that are not required in the management
of nonspecific reading disorders.
In the presence of significant reading
retarda-tion-usually two or more years below age-grade
level or mental age, although a retardation of
even one year may be regarded as diagnostically
significant-I make the diagnosis of
develop-mental dyslexia by analyzing reading and spelling
jointly as interdependent functions.’-2 My system
of analysis seeks to identify diagnostic
reading-spelling patterns not through errors alone, which
reflect only the dyslexic child’s functional deficits,
i.e., what he cannot do, but by means of the total
reading and spelling performance, which reflects
strengths as well as deficits.
It should be emphasized that my diagnostic
screening test is used in conjunction with the
main prevailing diagnostic criteria for
develop-mental dyslexia provided by (1) diagnosis by
exclusion, essentially a differential diagnosis, (2)
indirect diagnosis through neurological and
psychometric concomitants, and (3) direct
diag-nosis, based primarily on the frequency and
persistence of certain kinds of errors in reading
and writing. All of these standard approaches are
incorporated in the comprehensive diagnostic
work-up outlined in this paper.
The screening for atypical patterns begins with
the reading test, which consists of eight lists of 20
words each, both phonetic and nonphonetic, graded from pre-primer to sixth grade. These are
presented in two ways: “flash’ ‘ presentation
which determines the child’s reading level and
sight vocabulary, i.e., the words recognized
instantly as whole-word configurations, or
ge-stalts; and “untimed” presentation, which calls
upon the child’s ability to decode unfamiliar
words phonetically.
The spelling test, designed to parallel the
“flash” and “untimed” columns in the reading
test, is also given in two parts: “known” words
(
i.e., sight vocabulary) and “unknown” words (i.e.,not in sight vocabulary). A simple scoring
proce-dure is used, relating the child’s performance to
that of normal readers. It is based on the
percent-age of correctly spelled words in the “known”
word list, which is an index to the child’s ability to
“revisualize” words in his sight vocabulary, and
the percentage of “good phonetic equivalents” in
the “unknown” word list, which is an index to the
child’s word-analysis skills. (In the spelling
pattern of normal readers, 70% to 100% of
“known” words are spelled correctly; 80% to
100% of “unknown” words are spelled
phoneti-cally.’2)
My observations indicate that strengths and
deficits in the gestalt and analytic functions of
dyslexic children-the two basic processes
under-lying reading-are reflected in three atypical
reading-spelling patterns, none of which are
found among normal readers and spellers. On the
basis of these three patterns, three subtypes of
dyslexic children are delineated-dysphonetic,
dyseidetic, and mixed dysphonetic-dyseidetic,
each with its own prognostic and remedial
impli-cations. .2
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PEDIATRICS
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FIG. 1. Nonphonetic spelling in dysphonetic group 1. A 15-year-old boy, 10th grade, IQ 92 (Stanford-Binet LM). Reading level, fourth grade (sight vocabulary); word-analysis skills minimal even though he had been in a special reading class for three years. Known words: 25% correct, misspellings 67% dysphonetic. Unknown words: none correct, misspellings 100% dysphonetic. Note that some phonetic concepts are evident in his strikingly dysphonetic performance on the
unknown word list. (Reprinted with permission of Spastics International Medical Publications,
publishers of Developmental Medicine and Child Neurology.)
Croup 1 (dysphonetic), by far the largest of the
three subtypes, reflects a primary deficit in
symbol-sound integration, with inability to
devel-op word-analysis skills. These children read
globally, responding to whole words as
configura-tions, or gestalts. Lacking phonic skills, they are
unable to decode words not in their sight
vocabu-lary. Misspellings are typically nonphonetic and
unintelligible (Fig. 1). Their most striking errors,
primarily in reading but also in writing, are
semantic substitutions, e.g., reading “funny” for
“laugh” or “airplane” for “train.”
Croup 2 (dyseidetic) reflects a primary deficit
in the ability to perceive and recall whole words
as gestalts, without deficit in analytic function.
These children read primarily through a process
of phonetic analysis, sounding out most words as
if encountered for the firt time. Misspellings are
typically phonetic and intelligible (Fig. 2), e.g.,
“laf” for “laugh,” “lisn” fr “listen.”
Children who are both dysphonetic and
dysei-detic (Croup 3) are deficient both in the ability to
develop phonetic word-analysis skills and in
perceiving whole words as gestalts (Fig. 3).
Without remedial teaching, they tend to remain
virtually alexic-nonreaders and nonspellers.
ADDITIONAL DIAGNOSTIC STUDIES
At the conclusion of the first office visit, guided
by the review of past records, referrals for
addi-tional tests and diagnostic studies are made. The
regular consultants, described below, with each of
whom professional exchange by telephone is
readily available, constitute in effect a
multidisci-plmnary diagnostic team. The second office visit is
scheduled when reports on all the diagnostic
studies have been obtained.
Psychological Testing
Unless an adequate current report on
individ-ual psychometric testing is already available, the
child is referred to a clinical child psychologist for
a psychoeducational evaluation. When private
referral is not financially feasible, the cooperation
of the school psychologist can be invaluable. The
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FIG. 2. Phonetic spelling in dyseidetic group 2. A 10-year-old boy, fourth grade, IQ in bright normal range (Stanford-Binet LM), excels in arithmetic. Reading level, first grade (sight vocabulary); word analysis skills, third to fourth grade. Known words: 40% correct, misspellings 83% phonetic. Unknown words: none correct, misspellings 90% phonetic. Note that as in group 1 and 3 the total of correctly spelled words in the known words list is far below his reading
level.
evaluation is based on a minimum battery of
standard tests of intelligence and achievement,
supplemented with tests of visual and auditory
perception and memory and visuomotor
coordi-nation, and with projective tests, when possible,
to assess the emotional concomitants, primary
and secondary, of the child’s school failure.
Among the patterns of test performance upon
which I have come to rely for evidence of
cognitive deficits are certain characteristic WISC
profiles. These include a wide scatter in subtest
scores and a discrepancy of 15 IQ points or more
between the verbal and performance scales,
which are generally regarded as being diagnostic
of specific learning disabilities. A relatively low
verbal IQ and low scores on the digit-span and
coding subtests are especially significant in the
diagnosis of the genetic type of developmental
dyslexia, whereas a very low performance IQ is
frequently associated with overt neurological
evidence of brain damage.
Intelligence and achievement tests are key
instruments in assessing not only the degree of
underachievement but also the severity of a
specific learning disability. It is important to keep
in mind, however, that either the low
perform-ance IQ or the low verbal IQ and the wide subtest
scatter that are characteristic of children with
specific learning disabilities may bring their
frill-scale IQ on the WISC into the mentally retarded
range, rendering it meaningless as a measure of
overall intelligence and often leading to
misdiag-nosis and mismanagement.
A selective retardation in reading and spelling
on standard achievement tests, with normal
performance in arithmetic, justifying in such
cases the descriptive term “selective reading
disability,” is generally viewed as diagnostic of
developmental dyslexia. However,
developmen-tab dyslexia and dyscalculia can and frequently do
coexist in the same child. In my experience, it is a
reading-spelling discrepancy in achievement
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PEDIATRICS
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399
FIG. 3. Nonphonetic spelling of mixed dsphonetic-dyseidetic group 3. A 10-year-boy, fifth grade, IQ 87 (\VISC: verbal IQ 72, performance IQ 103). Reading level, pre-primer (sight
vocabulary); no word-analysis skills. Known words: 37% correct, misspellings 100% dysphonetic.
Unknown words: none correct, misspellings 100% dysphonetic. (Reprinted with permission of
Spastics International Medical Publications, publishers of Developmental Medicine and Child
Neurology.)
scores, with spelling at a much lower level, rather
than a reading-arithmetic discrepancy, that is the
more constant diagnostic sign of developmental dyslexia.
The visual memory, auditory memory, and
sound-blending subtests of the Illinois Test of
Psycholinguistic Abilities (ITPA) have proved to
be valuable supplements to my diagnostic
screen-ing procedure for developmental dyslexia, in that
they provide significant correlates with the three
identified dyslexic subtypes.
Additional Medical Diagnostic Studies
Since screening of vision and hearing in the
pediatrician’s office is usually inadequate for an
evaluation of a child with a learning disorder,
ophthalmobogical and oto-audiobogical
consulta-tions are routine, unless reports of recent
exami-nations are available.
The routine laboratory tests include a T, level
in addition to complete blood count and
urmnaly-sis, supplemented as indicated with selected
blood chemistries and special laboratory studies,
such as amino acid chromatography, cytogenetic
studies, skull X-ray films, and determination of
bone age. An EEC, taken during both the waking
and sleeping state, is obtained routinely on
chil-dren with intermittent inattention, severe
acting-out behavioral disorders, chronic brain
syn-dromes, mental retardation, or a history of seizures, but is not routine for children with
scholastic underachievement alone. Referral to a
speech pathologist or speech therapist is routine
for all children with speech impairment.
Psychiatric referral is made only for children
whose behavioral disorders suggest primary
emotional problems, borderline psychosis,
child-hood schizophrenia, or autism.
Since refractive errors that interfere with near
vision and reading comfort are easily missed in
routine visual screening, ophthalmobogical
evalu-ation with cycboplegic refraction is essential.
Although it is generally agreed that visual defects
are not a cause of specific reading disability, there
is a tendency to underestimate their importance
as a contributory factor or as a primary cause of
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nonspecific, secondary reading retardation.
Myo-pia, unless unusually severe, is not a factor in poor
reading, but even moderate hyperopia,
particu-larly if associated with astigmatism or
conver-gence insufficiency, may be a significant factor in
all reading disorders.
Oto-audiological evaluation is essential because
mid- and high-frequency hearing boss, which can
interfere with auditory discrimination sufficiently
to cause delay in speech and language
develop-ment, is usually missed except with pure-tone and
speech audiometry. Intermittent conductive
hy-poacusis due to recurrent or chronic serous otitis
mediacan also interfere with normal speech and language development and thereby result in a
secondary reading-spelling disability associated
with low scores on the WISC verbal scale similar
to those found in children with specific
language-learning disability.
TREATMENT
The multidisciplinary team approach essential
in the diagnosis of children presenting with school
failure is also the most effective approach to
treatment, provided that the pediatrician
contin-ues in the role of coordinator, implementing the
recommendations of the team and interpreting
them to the family and school personnel. The
recommendations must be coordinated into a
practical program of treatment in which all of the child’s multiple problems and correctable defects
are handled concurrently, taking into
considera-tion the whole child and the family’s actual
circumstances.
Despite the superficial similarities in the
symp-tomatology of children failing in school, each
child is found to have a distinctive cluster of
symptoms calling for a highly individualized
therapeutic approach. In my experience, unless
treatment is individualized, coordinated and
simultaneous, i.e., multifaceted, it is usually
unsuccessful. Drug therapy alone, for example, cannot take the place of needed educational
therapy
or psychotherapy. When a fullsimulta-neous treatment program is not feasible, realistic
priorities according to the specific immediate
needs of the given child have to be assigned.
Parent and child counseling is of paramount
importance in dealing with children who are
failing in school. The psychotherapeutic impact
of a correct diagnosis, particularly of
develop-mental dyslexia, can be quite dramatic. Leading
to an improved self-image for the child and to
better parent-child and teacher-child
relation-ships, the diagnosis itself often proves to be a
turning point in the child’s emotional
develop-ment and school adjustment, even when remedial
facilities are not immediately available.
Interpreting the dysfunction of central
pro-cesses essential for reading in terms of visual and
auditory “channels” for learning that are “open”
or “closed” is easily grasped and accepted by
parents and child. Furthermore, the child’s
reading-spelling patterns, being readily
demon-strable, can be used effectively to help parents
gain a better understanding of the child’s problem
and the goals toward which remedial instruction
is to be directed.
The special value of a structured home
environ-ment and good health habits for children with the
hyperkinetic syndrome or learning disabilities is
also stressed. It is felt that a structured home
routine helps the child to become better
orga-nized and to develop, along with better health
habits, better work habits and self-discipline, thus
contributing to a better school adjustment.
Communication With Schools
Utilization of school personnel as an integral
part of the multidisciplinary team is especially
advantageous. Once the child’s behavioral and
learning problems have been properly diagnosed
andinterpreted to key school personnel, they can
begin to share in the management.
Awareness of community resources, both
public and private, enables the pediatrician to
guide the parents in obtaining the best and most
practical program available for their child.
School structuring or special education
pro-grams are indicated for most children with the
hyperkmnetic syndrome or learning disabilities.
For mild or moderate specific learning
disabili-ties, just as for nonspecific ones, school
structur-ing may suffice. Immediate classroom adaptations
to make allowances for the child’s limitations
include such simple measures as not requiring
oral reading and spelling before the class when
the child’s problem is one of dyslexia, giving oral
rather than written tests and providing
oppor-tunity for activities in which the child can
succeed. A year’s retention may be a valuable
adaptive measure at kindergarten or first-grade
level, though usually not later. Whenever
possi-ble, it is advisable to have the child remain in a
regular classroom, supplemented with the
appro-priate remedial programs at school or through
private
tutoring.A full-time learning disability class within the
schools is recommended for children with severe
learning disabilities and for those who are not
making satisfactory progress in a part-time
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dial program. A specialized private school, for
which state funds may be obtainable, is indicated
for children with severe learning disabilities for
whom no effective public school program is
available.
In addition, children with general motor
clum-siness, who experience frustration on the
play-ground, can often be helped in a corrective
physical education class at school; children with
speech impairments or poor auditory
discrimina-tion may be able to benefit from the school’s
speech therapy program.
Educational Therapy
Special education is indispensible in the
reme-diation of specific learning disabilities. No other
measures, alone or in combination, can take its
place.
It is not, however, within the scope of this
paper to review the variety of remedial
tech-niques utilized by educators, although the
pedia-trician interested in learning disabilities should
have some familiarity with them. In general, the
techniques are multisensory, i.e., visual, auditory,
and tactile-kinesthetic, and often addressed
primarily to one sensory modality and reinforced
by another.
Although opinions differ on whether to teach
initially to the “open” or the “closed” channel,
that is, to the strength or to the weakness, my own
experience with the three dyslexic subtypes
supports the view that the initial approach should
be to teach to the strength, not only to ensure
initial successes for the child but also to prepare a
better foundation for teaching to the weakness
with remedial techniques.
Psychotropic Drug Therapy
Since hyperkinetic children are a
heteroge-neous group, only a careful differential diagnosis
can serve as a basis for determining whether drug
therapy is indicated.
The basic distinction is between neurogenic
hyperactivity, i.e., the classic hyperkinetic
syndrome, and psychogenic hyperactivity, i.e., a
behavioral response to anxiety. Though
psycho-stimulants are the drugs of choice for neurogenic
hyperactivity, tranquilizers or tricyclic
antide-pressants may be indicated for psychogenic
hyperactivity. When a diagnosis of hyperkinetic
syndrome is in doubt, a diagnostic trial of
psychostimulants is justified.
When a child with the hyperkinetic syndrome
has severe emotional concomitants, notably poor
self-image with depression or acting-out behavior,
a psychostimulant may be combined with a
tranquilizer or tricyclic antidepressant,
prefer-ably in conjunction with psychotherapy.
In mild developmental hyperactivity,
psycho-stimulants are not indicated; home structuring
and behavior modification techniques at home
and at school usually suffice. When the
hyperki-netic syndrome is associated with a specific
learn-ing disability, psychostimulants may be successful
initially in modifying the child’s school behavior
only to be followed by a recurrence of
hyperkmne-sis, the neurogenic hyperactivity being replaced
by anxiety-based hyperactivity. Thus, when
psychostimulants fail to ameliorate a true
hyper-kinetic syndrome, a coexisting specific learning
disability or a primary emotional problem must
be considered. A nonspecific learning disability
secondary to attentional deficit and hyperkinesis
usually resolves spontaneously with successful
psychostimulant therapy without special
educa-tion or individual tutoring.
In helping parents to accept a trial of
psycho-stimulant therapy, it is important to explain that
the objective is not to sedate or subdue the child
but rather to “normalize” his behavior, overcome
the attentional deficit, thus making the child
more accessible to learning, and improve school
adjustment. It is also helpful to explain that
developmental hyperactivity tends to diminish
with maturation, and that the medication can be
discontinued when the child develops better
ability to concentrate and achieves adequate
impulse control. Care is taken that the child on
medication is not deprived of a sense of
accomplishment for improved behavior by
point-ing out to him that medication can help his
behavior and learning only if he himself makes
the effort to improve.
When medication is prescribed, the need for
adult supervision is emphasized. Large dosages
are avoided because of their side effects. When
moderate dosage is not effective, trial of a
different medication is made. Anorexia and
insomnia, the most common side effects of
psychostimulants, may be minimized by limiting
the medication to the morning and noontime. In
selected cases in which the attentional deficit and
hyperkinesis create problems primarily at school,
I have found it reassuring to parents to suggest
that medication may be discontinued on
weekends and during school vacations.
Psychotherapy
Psychotherapy is usually not required for the
secondary, reactive emotional overlay invariably
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present in the child failing in school. In my
experience, like that of others, the overlay tends
to
clear up once the underlying problems havebeen managed medically and educationally. In
older children, however, the emotional
concomi-tants of chronic school failure are frequently so
severe that referral to a child psychiatrist or
clinical psychologist experienced in dealing with
school problems or to a child guidance clinic
should be considered.
If parent counseling and behavior therapy do
not resolve the child’s primary or secondary
emotional and motivational problems, individual
psychotherapy or family therapy is indicated.
Croup therapy is usually preferable for
adoles-cents. Parents may be encouraged to participate
in an effective parent organization such as the
Association for Children with Learning
Disabili-ties (ACLD), as a means of increasing their insight
into the child’s problems and allaying their own
anxiety.
Follow-up
Follow-up on academic progress and the
effec-tiveness of psychotropic drug therapy, both
requiring close communication with school
personnel, is viewed as an essential aspect of
management. Before each office visit, a simple
questionnaire on behavior and achievement is
mailed directly to the school, so that a
confiden-tial report from the teacher, which is
indispens-ible as a guide to adjusting psychostimulant
medication, will be available.
The best evidence for the effectiveness of
management is relatively rapid behavioral or
academic improvement within the current
semes-ter or school year, especially if the child’s
problem has been chronic and has failed to
respond to previous therapeutic measures. Other
criteria for evaluating management include
improved parental attitudes toward the child and
the school, and improvement in the child’s
self-image and attitude toward school.
In assessing the effectiveness of a given dosage
of psychostimulant, I supplement the parents’ and
teacher’s reports periodically with an objective
before-and-after office test, for which the parents
are asked to bring the child to the office without
medication. A set of paper-pencil tasks is given.
This includes copying of geometric designs,
drawing the child’s own family, and writing to
dictation a selected list of words from the
diag-nostic screening procedure for developmental
dyslexia. The previously established dosage of
psychostimubantt is then administered; 30
mm-utes later the paper-pencil tasks are repeated.
When psychostimulant therapy is effective,
strik-mg improvement in test performance is usually
noted. Handwriting and drawings tend to be
smaller and neater, and hyperactivity and
dis-tractibility are diminished. For a quantitative
assessment of the effectiveness of the
psychostim-ulant, the Coodenough Draw-a-Person-Test and
the Bender Cestalt Test may be given as part of
the before-and-after office test and scored later by
a clinical psychologist.
For children receiving psychostimulant
medi-cation, it is important to follow the height and
weight curves, since anorexia and insomnia, the
most common side effects, may interfere with
so-matic growth.
SUMMARY AND CONCLUSIONS
A comprehensive multidisciplinary team
ap-proach
to the diagnosis and management ofschool failure, with the pediatrician
knowledge-able in the field of behavioral and learning
disorders serving as coordinator, is outlined in
terms of actual office procedures. An integral part
of the expanded neurological examination is an
empirically evolved diagnostic screening test for
developmental dyslexia which identifies three
atypical reading-spelling patterns on the basis of
which dyslexic children can be classified into
three main subtypes: dysphonetic, dyseidetic, and
mixed dysphonetic-dyseidetic, each with its own
therapeutic and prognostic implications.
The approach outlined here is designed to
identify not only the specific educational needs of
a given child but all of the causative and
contrib-utory factors, physical, emotional, and
sociocultu-ral, that may impinge on the child’s ability to
learn. Though necessarily time-consuming, the
team approach appears in our present state of
knowledge to be the most practical and reliable
way to get at the roots of the child’s multifaceted
problem and to develop a coordinated program of
treatment-simultaneous insofar as feasible, and
sequential when required.
A child referred for school failure is a child in
crisis. The challenge to the pediatrician to whom
tSee Denhoff’s article in this issue for an outline of psycho-stimulant medication dosages.
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PEDIATRICS
Vol. 58 No. 3
September 1976 403the parents turn for a solution is to find ways as
promptly as possible to help the child improve in
his school adjustment and self-esteem. It is
recog-nized that special education has a primary and
ultimate responsibility for remediating specific
learning disabilities. Nevertheless, the complexity
of the problem of school failure requires that
overall diagnosis and management be a
multidis-ciplmnary venture, in which the professionals
involved
are committed to working together toensure that the whole child is taken into
account.
REFERENCES
1. Boder E: Developmental dyslexia: Prevailing diagnostic concepts and anew diagnostic approach. In, Mykle-bust HR (ed): Progress in Learning Disabilities:
Volume II. New York, Grune & Stratton, 1971. 2. Boder E: Developmental dyslexia: A diagnostic approach based on three atypical reading-spelling patterns. Dev Med Child Neurol 15:663, 1973.
The
Assessment
of the
Child
at School
Entry
Martin C. 0. Bax, M.B.
From the Thomas Coran, Research Unit, London University, London, England
Most learning and behavior disorders in school
children present as “crisis situations,” when
anxious parents or exasperated teachers suddenly
call for medical or psychological help with a child
aged 9, 10, or 11 who is in severe difficulties. It is
not surprising that remedial efforts to help these
children are often unsuccessful. It seems likely
that if we could identify the children earlier and
try to help them before they get into difficulties,
the results would be much better. However, the
correlation between abnormalities found in
babies in the newborn period or in the early years
of life, although significant, are quite low, and we
cannot identify with any degree of reliability
children who may have school difficulties at this
earlier age. On the other hand, we have shown
that a thorough assessment of the child at school
entry will allow one to identify quite a large
proportion of the children who are going to get
into difficulties.’ A careful assessment, therefore,
of children at school entry is an essential part of
educational pediatrics.
There are a number of other reasons why
preschool assessments cannot identify potential
problems. The main one is that there are a
number of functions emerging about age 5 or 6
which cannot be adequately studied in the early
years. A further, and very important, reason for
putting a great deal of emphasis on the initial
examination at school is that whatever
adminis-trative arrangements, under the various systems
of medicine that operate in different countries,
are made in the preschool period to see children,
it is difficult to be certain of seeing all the
children. It is just those children who are not
brought by their parents for regular preschool
ADDRESS FOR REPRINTS: Thomas Coram Research Unit,
University of London, London, England.
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1976;58;394
Pediatrics
Elena Boder
Evaluation and Treatment
−−
School Failure
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1976;58;394
Pediatrics
Elena Boder
Evaluation and Treatment
−−
School Failure
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