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Inhibition of receptor localized PI3K preserves cardiac β adrenergic receptor function and ameliorates pressure overload heart failure

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Figure

Figure 1Generation of transgenic mice with decreased cardiac phatidylinositol mono-phosphate; PIP2, phosphatidylinositol PI3Kpholipase Cimmunoprecipitation; associated PI3K activity in the left LVs measured by their ability to phosphorylate PtdIns-4,5-Ptra
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