The PTPN14 Tumor Suppressor Is a Degradation Target of Human Papillomavirus E7

HeLa cells stably expressing huTRIM5 ␣ or rhTRIM5 ␣ were transfected with siRNAs against DHC (siDHC) or against luciferase (siLuc) as a control and infected 72 h later in

Therefore, CaSKi cells were transfected with E2 and the levels of E7 protein in a total cell extract were analyzed by Western blotting with antibodies against E7 and actin as a

Huh-7 cells were transfected with pHDV 䡠 I( ⫹ ) and the indicated siRNAs; RNAs were harvested 6 days posttransfection and analyzed for HDV amber/W editing as in Fig.. 4)

To assess whether the expression of Nef during HIV-1 infection reduces the levels of endogenous p53 and inhibits the induction of p53 by cells in response to UV irradiation, extracts

p53 immunoprecipitations from the rare colonies of cells arising following transfection with pE7 plus ras plus wild-type p53 or Ela plus ras plus wild-type p53 show that the level

Overexpression of SOX9 in cervical carcinoma cells (SiHa and C33A) inhibited cell growth in vitro and tumor formation in vivo.. In agreement, the silencing of SOX9 in HeLa

p53; human papilloma virus; cervical carcinoma; HeLa cells; chemical library; transcriptional reporter construct; β -galactosidase; suppression of cell proliferation;

Confirmation of the expression of HPV type 16 E6 protein and scanning the expression of 11 host miRNAs in Hela cells: (A) E6 protein was only detected in E6 expression vector