Evidence for a group specific enteroviral antigen(s) recognized by human T cells

To determine the feasibility of developing a murine model, we previ- ously subcloned the human a-1,2-fucosyltransferase (H-transferase, EC 2.4.1.69) cDNA and the human

In summary, the results described herein suggest that genetic variation and IFN-I signaling determine HBV-specific CD8 ⫹ T cell responses by regulating the initial antigen

These EBV-LCL J1-stimulated T lymphocytes act as antigen-specific regulatory cells, since their addition to fresh autologous T cells cultured with autologous nontransduced EBV-LCL

Most of the tumor antigens described thus far have relied on the definition of targets recognized either by T cells infiltrating into cancer tissue or from T cells harvested

Further, in contrast to earlier suggestions, tat expression in antigen-specific T-cell clones at levels sufficient to support active HIV-1 production did not appreciably alter

The antigen (henceforth called C33 antigen) was about 40 to 55 kDa in HTLV-1-negative Jurkat, CEM, MOLT-4, and normal peripheral blood CD4-positive human T cells and about 40 to 75

Lack of relationship between virus-specific surface and transplantation antigens in hamster cells trans- formed by simian papovavirus SV40. New surface antigen in cells transformed

Although the frequency of antigen-specific CD4 + T cells in human peripheral blood appears to be below the limit of direct multimer staining, these reagents, in conjunction with