HEP-G2 liver cells
Nielsen, Carina (2009): Untersuchungen zur Toxizität und zu den molekularen Wirkungsmechanismen von Deoxynivalenol. Dissertation, LMU München: Tierärztliche Fakultät
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Journal of Applied Pharmaceutical Science
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Suppression of E-cadherin Mediates Gallotannin Induced Apoptosis in Hep G2 Hepatocelluar Carcinoma Cells
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BISPHENOL A ENHANCES GROWTH OF Hep G2 CANCER CELLS BY UPREGULATING EXPRESSION OF PRO – INFLAMMATORY AND PRO – ANGIOGENESIS PROTEINS
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Evaluation of pullulan-functionalized doxorubicin nanoparticles for asialoglycoprotein receptor-mediated uptake in Hep G2 cell line
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Synthesis, Characterization and in vitro Cytotoxic Evaluation of Some Novel Heterocyclic Compounds Bearing the Indole Ring
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Acacetin inhibits the proliferation of Hep G2 by blocking cell cycle progression and inducing apoptosis
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THE ANTIOXIDANT ACTIVITY AND CYTOTOXIC ACTIVITIES AGAINST CANCER CELL LINES OF PROMPAK REMEDY
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IN VITRO CYTOTOXIC ACTIVITY OF SQUID AND CUTTLEFISH BONE EXTRACT ON HEP G2 CELL LINE
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Targeting DTL induces cell cycle arrest and senescence and suppresses cell growth and colony formation through TPX2 inhibition in human hepatocellular carcinoma cells
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Characterization of precursor and secreted forms of human angiotensinogen
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Ultrasonic assisted synthesis, anticancer and antioxidant activity of somenovel pyrazolo[3,4 b]pyridine derivatives
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Combined incubation of Cadmium, docosahexaenoic and eicosapentaenoic acid results in increased uptake of cadmium and elevated docosapentaenoic acid content in Hepatocytes in vitro
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Regulation of sex hormone binding globulin and insulin-like growth factor binding protein-1
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“Evaluation of Anticancer Activity of Mazus pumilus Leaf Extracts on Selected Human Cancerous Cell Lines” by Pittu Vishnu Priya, Avanapu Srinivasa Rao, India.
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Lrig1 is a positive prognostic marker in hepatocellular carcinoma
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UBQLN4 promotes progression of HCC via activating wnt-β-catenin pathway and is regulated by miR-370
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Effects of Caffeine Againts Expression on Mir-423-3p in Cell Lines Hep-G2
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Adverse effects of anti tuberculosis drugs on HepG2 cell bioenergetics
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