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ABSOLUTE CONTRAINDICATIONS FOR EXERCISE Depending on any other co-morbidities absolute

In document Exercise Prescription - eBook (Page 62-68)

RESISTANCE CIRCUIT TRAINING AND WEIGHT TRAINING

ABSOLUTE CONTRAINDICATIONS FOR EXERCISE Depending on any other co-morbidities absolute

con-traindications for exercise will be as for those cited for other individuals (Appendix A, Table A.1)

Suggested Readings, References and Bibliography 51

Suggested readings, references and bibliography

Angelopoulos T J, Robertson R J 1993 Effect of a single exercise bout on serum triglyceride in untrained men. Journal of Sports Medicine and Physical Fitness 33:264–267

Angelopoulos T J, Robertson R J, Goss F L et al 1993 Effect of repeated exercise bouts on high density lipoprotein-cholesterol and its subfractions HDL2-C and HDL3-C. International Journal of Sports Medicine 14(4):196–201

Aronov D M, Bubnova M G, Perova N V et al 2003 Physical exercise and atherosclerosis: proatherogenic effects of high and moderate intensity static exercise on blood lipid transport. Kardiologiia 43(2):35–39

Assmann G 1982 Lipid metabolism and atherosclerosis. Schattauer Verlag, Stuttgart

Ballantyne F C, Clark R S, Simpson H S et al 1982 The effect of moderate physical exercise on the plasma lipoprotein

subfractions in male survivors of myocardial infarction. Circulation 65:913–918

Barletta C, Coiloi I, Barlolomei D I et al 1993 Influence of aerobic physical activity on blood lipids in relation to sex and training.

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Pharmaceutical Press, London

Suggested reading

Assmann G 1982 Lipid metabolism and atherosclerosis. Schattauer Verlag, Stuttgart. (In-depth description of the various

hyperlipidaemias and dyslipidaemias.)

Miller N E, Miller G J 1984 Clinical and metabolic aspects of high-density lipoproteins. Elsevier, Amsterdam

Thompson G R 1989 A handbook of hyperlipidaemia. Current Science, London. (In-depth description of the various hyper- and

dyslipidaemias.)

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SUMMARY

Hyperlipidaemia is characterized by increased levels of serum TGs, cholesterol and fatty acids, and is a primary risk factor for heart and vascular disease.

CHD rises appreciably when serum cholesterol is

>6.5 mmol/l (250 mg/dl), and more so when

>7.8 mmol/l (300 mg/dl). Lowest rates occur in men with serum cholesterol ≤5.2 mmol/l (200 mg/dl).

LDL-C >3.36 mmol/l (130 mg/dl) is a risk to health and generally deposits some cholesterol into arterial walls.

Compared to premenopausal women, men tend to have a poorer BLP. After menopause the gender difference tends to diminish.

Regular PA/exercise reduces TG and increases

HDL-C levels. HDL-Changes appear dependent on PA/exercise intensity, duration and total energy expenditure, and baseline blood lipid levels.

Regular energy expenditure through physical activity of around 1000 kcal per week is required to induce improvements in HDL-C.

Habitual aerobic PA/exercise appears more effective than strength or cross training in producing favour-able changes in BLP in healthy, previously sedentary adults.

Regular resistance training has been shown to pro-duce improvements in BLP.

Transient changes in TG, HDL and apo A can result from a single bout of PA/exercise, through the action of LPL. The magnitude appears dependent on train-ing status, duration, intensity and energy expen-diture of the PA/exercise bout.

Individuals with familial dyslipidaemia/s may not respond to PA/exercise intervention in the same way as individuals suffering from secondary dyslip-idaemia. However, other potential health benefits may be gained.

The PA/exercise-induced changes in BLP tend to diminish within several weeks from the cessation of training.

Medications prescribed for dyslipidaemic and hyperlipidaemic individuals may affect PA/exercise capacity.

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