Functional characterization of a U5 ribozyme: intracellular suppression of human immunodeficiency virus type 1 expression.

To investigate the protective effect of the CCR2 polymor- phism, we studied the expression as well as the chemokine receptor and HIV-1 coreceptor activities of the major CCR2

These data imply that activa- tion of the cell cycle in primary epithelial cells by E1A is sufficient to allow efficient oncogenic transformation of such cells by v-src and that

Our data also demonstrate a novel link between type A retroelements and type D retroviruses since both con- tain functional posttranscriptional control elements that share

Similar reports of HCMV-induced cellular DNA replication by others (4, 6, 13, 36, 39), coupled with the observations that HCMV infection can induce several enzymes that function in

All the above data suggest that HIV infection of macrophages results in the mod- ification of signal transduction pathways and transcription fac- tors to create a favorable

For those CD4- positive cells from infected cultures that were stained positively with anti-HIV MAbs, staining was weakest with anti-CD4bd MAbs, intermediate with anti-gp41 and

recognized by lesion-infiltrating HSV-specific CD4+ T cells provides a minimum estimate of the diversity of viral epitopes because of the small number of patients studied, the

(43, 44) reported the role of Ca2+ in the in vitro assembly of murine polyomavirus recombinant VP1 capsomeres into capsid-like particles, and now we have reported a similar mechanism