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Caveolin-1 contributes to realgar nanoparticle therapy in human chronic myelogenous leukemia K562 cells

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Figure 1 realgar NPs induced cell death and fusion protein degradation.Notes: (A) K562 cells were seeded in 96-well plates and treated with specific concentrations of realgar NPs (0, 5, 10, 20, 40, 80, and 100 μg/ml) for 24, 48, or 72 h
Figure 2 realgar NPs activated apoptosis in K562 cells.Notes: (A) Cells were treated with specific concentrations of realgar NPs (a: 0 μg/ml; b: 5 μg/ml; and c: 10 μg/ml) for 24 or 48 h (d: 5 μg/ml and e: 10 μg/ml) and stained by annexin V and PI staining
Figure 3 realgar NPs triggered autophagy in K562 cells.Notes: (A) The cells were treated with specific concentrations of realgar NPs (0, 5, and 10 μg/mL) for 24 h and then fixed and immunostained with antibodies against LC3 (green), and nuclei were counter
Figure 4 realgar NPs activated autophagy through PI3K/akt/mTOr pathway.Notes: (A) K562 cells were treated with specific concentrations of realgar NPs (0, 5, 10, and 20 μg/ml) for 24 h
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