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Protective effects of NAT-CLW TM on acetaminophen-induced oxidative stress-mediated apoptosis in hepatic HepG2 cells

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Figure

Figure 1: Displaying the MTT cell viability analysis of silymarin and NAT-CLW in the APAP-induced HepG2 cells
Figure 2: (a-c) Effect of NAT-CLW and silymarin on TBARS, GSH levels, and SOD enzyme activities in APAP-induced HepG2 cells
Figure 3 shows a significant increase an intracellular ROS  formation in APAP (20 mM) induced in HepG2 cells as compared  to control cells

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