Lipid Hydroperoxide
Presence of lipid hydroperoxide in human plasma
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The Effect of N-acetylcysteine on Pyrrolized Protein, Lipid Hydroperoxide and Thiol Levels in the Carbon Tetrachloride Hepatotoxicity
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The Relation of Lumbar Disc Herniation With Increased Lipid Hydroperoxide, Paraoxonase 1 and Total Oxidative Status
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In vitro cell injury by oxidized low density lipoprotein involves lipid hydroperoxide induced formation of alkoxyl, lipid, and peroxyl radicals
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Advanced platform for shelf life extension in liquid foods : a thesis presented in partial fulfillment of the requirements for the degree of Doctor of Philosophy in Bioprocess Engineering at Massey University, Palmerston North, New Zealand
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Title: ADAPTIVE IMMUNE SYSTEM: FRIEND OR FOE? Author: Mr. Anoop Kumar Keyword: Guest EditorialPage No: iAbstract: Adaptive immune system: Friend or Foe? Immunity is body’s self defense mechanism for protecting the body from infectious diseases or other antigen attacks. The immune system is a tightly regulated co mplex network, including lymphoid, reticular, dendrite and epithelial cells, interacting by cell to cell contacts and communicating via soluble mediators such as cytokines. Adaptive immune system is a more heterogeneous and complex system comprising of T and B cells. Adaptive responses are generated in the lymph nodes, spleen and mucosa-associated lymphoid tissue. It involves the proliferation of antigen-specific B and T cells. Specialized cells, called antigen-presenting cells, display the antigen to lymphocytes and collaborate with them in response to antigen. B cells secrete immunoglobulins (IgG) and T cells help B cells to make antibody. Antigen recognition is major histocompatibility complex (MHC) restricted. Immune memory also is a hallmark feature of the adaptive immune system and results in a more efficient response upon secondary exposure to the same antigen. So, the adaptive immune system acts as our friend to combat against pathogens but the mechanism by which T and B cell protect our body against pathogens is still unclear. For better understanding of its mechanism, it should be explores in future studies. When this system unable to recognize its own cell, it become our enemy which results in various autoimmune disorders such as rheumatoid arthritis, type 1 DM, systemic lupus erthromatous etc. In literature various studies has been conducted to understand their mechanism but unfortunately how the adaptive immune system become our foe is still unclear. So, In future studies, researchers should plan their research to explore this issues which will be helpful to design drugs and vaccines for autoimmune disorders. Download PDF
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7,8 Dihydroneopterin mediated protection of low density lipoprotein, but not human macrophages, from oxidative stress
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Formation of (2E) 4 Hydroxy 2 nonenal and (2E) 4 Hydroxy 2 hexenal by Plant Enzymes: A Review Suggests a Role in the Physiology of Plants
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Biliary glutathione promotes the mucosal metabolism of luminal peroxidized lipids by rat small intestine in vivo
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<em>Acinetobacter baumannii</em> biofilms: effects of physicochemical factors, virulence, antibiotic resistance determinants, gene regulation, and future antimicrobial treatments
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Effects of hypo and hyperoxia on transcription levels of five stress genes and the glutathione system in liver of Atlantic cod Gadus morhua
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Oxidative Stress in Female Athletes Using Combined Oral Contraceptives
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Early detection of multidrug and pre extensively drug resistant tuberculosis from smear positive sputum by direct sequencing
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Fatty acid oxidation products (‘green odour’) released from perennial ryegrass following biotic and abiotic stress, potentially have antimicrobial properties against the rumen microbiota resulting in decreased biohydrogenation
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Antimutagenic and DNA Damage Protective Activities of a Grape Extract from Vitis vinifera
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Synthesis of [alpha] farnesene autoxidation products and cross conjugated polyenes : presented in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Chemistry at Massey University
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Mimicking of glutathione peroxidase deficiency by exposition of JAR cells to increased level of synthetic hydroperoxide
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Intramolecular Hydrogen-shift Reactions of Peroxy Radicals
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Mimicking ClassIb Mn<inf>2</inf> Ribonucleotide Reductase: A Mn<sup>II</sup><inf>2</inf>Complex and Its Reaction with Superoxide
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