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[PDF] Top 20 Alzheimer's Disease: Synaptic Dysfunction and Aβ

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Alzheimer's Disease: Synaptic Dysfunction and Aβ

Alzheimer's Disease: Synaptic Dysfunction and Aβ

... (dodecamer) species were detected at an age that coincided with the first observed changes in spatial memory ...[86]. monomer, trimer and hexamer were seen at earlier time points and hence were not ... See full document

14

Oligomeric Aβ-induced synaptic dysfunction in Alzheimer’s disease

Oligomeric Aβ-induced synaptic dysfunction in Alzheimer’s disease

... ’ s disease (AD) is a devastating disease characterized by synaptic and neuronal loss in the ...induce synaptic loss in AD. A β -induced synaptic dysfunction is dependent ... See full document

12

Decoding the synaptic dysfunction of bioactive human AD brain soluble Aβ to inspire novel therapeutic avenues for Alzheimer’s disease

Decoding the synaptic dysfunction of bioactive human AD brain soluble Aβ to inspire novel therapeutic avenues for Alzheimer’s disease

... of on synaptic plasticity, we used several epitope-specific ...neutralize -mediated synaptotoxicity and reduce plaque load [3, 64, 83], thus leading to most anti- antibodies ... See full document

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ALZHEIMER’S DISEASE: A REVIEW

ALZHEIMER’S DISEASE: A REVIEW

... of . Furthermore, the development of Alzheimer disease in individuals with trisomy 21 has been related to a gene dosage effect with increased production of APP and subsequently ...familial ... See full document

9

Neurogranin and YKL-40: independent markers of synaptic degeneration and neuroinflammation in Alzheimer’s disease

Neurogranin and YKL-40: independent markers of synaptic degeneration and neuroinflammation in Alzheimer’s disease

... The study protocol was approved by the ethics commit- tee of the university clinic Erlangen-Nürnberg (number 3987), and all participants provided written informed consent. The patients were recruited in the memory clinic ... See full document

8

Dominantly Inherited Alzheimer Network: facilitating research and clinical trials

Dominantly Inherited Alzheimer Network: facilitating research and clinical trials

... CSF assays) as well as future ...National Alzheimers Coordinating Center, the National Cell Repository for Alzheimer’s Disease, and the Alzheimers Disease ... See full document

7

Mitochondrial β amyloid in Alzheimer's disease

Mitochondrial β amyloid in Alzheimer's disease

... intracellular -binding protein and was first identified in 1997 using a yeast two-hybrid screen ...with (33). However the precise residues involved in the -ABAD interaction could not be ... See full document

11

Alzheimer’s Disease Assessment Scale–Cognitive subscale variants in mild cognitive impairment and mild Alzheimer’s disease: change over time and the effect of enrichment strategies

Alzheimer’s Disease Assessment Scale–Cognitive subscale variants in mild cognitive impairment and mild Alzheimer’s disease: change over time and the effect of enrichment strategies

... In the context of this study, we also sought to deter- mine whether enrichment strategies could improve the sensitivity to change over time of these ADAS-Cog vari- ants. Since the introduction of enrichment as part of ... See full document

13

Proteinopathy, oxidative stress and mitochondrial dysfunction: cross talk in Alzheimer's disease and Parkinson's disease

Proteinopathy, oxidative stress and mitochondrial dysfunction: cross talk in Alzheimer's disease and Parkinson's disease

... Alzheimer’s disease and Parkinson’s disease are two common neurodegenerative diseases of the elderly people that have devastating effects in terms of morbidity and ...the disease in either case is ... See full document

14

Genome-wide association study for variants that modulate relationships between cerebrospinal fluid amyloid-beta 42, tau, and p-tau levels

Genome-wide association study for variants that modulate relationships between cerebrospinal fluid amyloid-beta 42, tau, and p-tau levels

... Alzheimer’s disease (AD). Cerebrospinal fluid (CSF) levels of amyloid-beta () and tau change before clin- ical symptoms of AD are ...the disease progresses, and tau levels ... See full document

9

Untangling the Tauopathy for Alzheimer’s disease and parkinsonism

Untangling the Tauopathy for Alzheimer’s disease and parkinsonism

... Although AD and PD are considered as diseases prevalent in the elderly, the initiation of neuron pathology associated with tauopathy in the AD, PD, and other tauopathy-associ- ated neurodegenerative diseases starts early ... See full document

11

Cognitive reserve and clinical progression in Alzheimer diseaseA paradoxical relationship

Cognitive reserve and clinical progression in Alzheimer diseaseA paradoxical relationship

... (median 12 months), possibly because repeated cognitive testing is more challenging at clinically advanced stages. As cognitive changes may be harder to capture over shorter follow-up durations, this could have caused a ... See full document

14

Design and first baseline data of the DZNE multicenter observational study on predementia Alzheimer’s disease (DELCODE)

Design and first baseline data of the DZNE multicenter observational study on predementia Alzheimer’s disease (DELCODE)

... Memory services are often approached by individuals who subjectively experience decline in cognitive func- tioning but perform within the age, sex, and education- adjusted normal limits on standard cognitive tests. In- ... See full document

10

Alzheimer’s in a dish – induced pluripotent stem cell based disease modeling

Alzheimer’s in a dish – induced pluripotent stem cell based disease modeling

... pathology has been described to interfere with synaptic processes and mediate synaptic degener- ation and toxicity (reviewed in ...of synaptic function and ...of to healthy ... See full document

13

Baicalein improves behavioral dysfunction induced by Alzheimer’s disease in rats

Baicalein improves behavioral dysfunction induced by Alzheimer’s disease in rats

... II: disease control treated with AlCl 3 to induce the mimicking AD for 4 successive weeks (SW); Group III: normal control group treated with baicalein (5 mg/kg) for 2 SW followed by combination of baicalein and ... See full document

8

The Evolution of Aβ Peptide Burden in the APP23 Transgenic Mice: Implications for Aβ Deposition in Alzheimer Disease

The Evolution of Aβ Peptide Burden in the APP23 Transgenic Mice: Implications for Aβ Deposition in Alzheimer Disease

... The heterogeneity exhibited in almost every as- pect of AD patients has complicated attempts to de- fine risk factors and predict the course of the disease. Consideration of the incomplete analogy between AD and ... See full document

10

Cholinergic dysfunction in a mouse model 
of Alzheimer disease is reversed 
by an anti Aβ antibody

Cholinergic dysfunction in a mouse model of Alzheimer disease is reversed by an anti Aβ antibody

... The peptide was coimmunoprecipitated from hippocampal extracts prepared from PDAPP trans- genic mice by an anti–ChT-1 antibody followed by Western blot analysis with bioti- nylated anti- antibodies ... See full document

9

The role of cerebral amyloid β accumulation in common forms of Alzheimer disease

The role of cerebral amyloid β accumulation in common forms of Alzheimer disease

... and generation ...of by stimulating α-secretase to yield a processing pattern that is indistinguishable from regulated cleavage modulated by PKC and MAPK/ERK ...brain accumulation ... See full document

10

Olfactory dysfunction in Alzheimer’s disease

Olfactory dysfunction in Alzheimer’s disease

... olfactory dysfunction caused by diseases. It is also unclear during which disease stage AD pathological changes are limited to the olfactory ...olfactory dysfunction and other biomarkers of AD remain ... See full document

7

Prevalence of preclinical Alzheimer diseaseComparison of current classification systems

Prevalence of preclinical Alzheimer diseaseComparison of current classification systems

... Among the strengths of this study are the representative population-based sample, the relatively high response rate for LP, and the comprehensive examinations conducted by trained psychiatric nurses. Some limitations ... See full document

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