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Status Epilapticus

Status Asthamaticus

Group members Shazia,valentina,varda, uzma

At SNC Lahore

(3)

Presentation Plan

Subject: ACCN Class PRN BSN Topic: Status Epilapticus and Date: 01-06-2017 Status Asthmaticus

Mentor: Tanzeel Ul Rahman

Sr No Objectives Content Time Strategies/Discussion Evaluation

1

Explore status epilepticus

Status Asthamaticus

Definition

Difference R/T epilapsy

and asthama

5

Multimedia Discussion

Question &

Answers

2

Its pathophysiology

Etiology and pathogenesis,

10

Multimedia Discussion

Question &

Answers

3

Clinical manifestation

Sign and symptoms

5

Multimedia Discussion

Question &

Answers

4

Medical Management

Surgical management

Initial Management

10

Multimedia Discussion

Question &

Answers

5

Explanation of Nursing

diagnosis and

Management

Nursing diagnosis and

Management

10

Multimedia Discussion

Question &

Answers

(4)

Learning Outcomes/Objectives

What is status epilepticus and Status Asthamaticus

Pathophysiology

Etiology

Pathogenesis

Clinical Manifestations

o

Signs

o

Symptoms

Medical Management

Surgical Management

(5)

Status Epilepticus

 Status Epilepticus

It is defined as continuous clinical and electrographic seizures activity. A seizure

that longer than 5 minutes, or having more than 1 seizure with in a 5 minutes period, without returning to a normal level of consciousness between episodes is called status epilepticus. This definition used a 30 minute time limit.

Convulsions may involve jerking motions, grunting sound, drooling and rapid eye

movements . The seizures can either be of the tonic-clonic types with regular

pattern of contraction and extension of the arms and legs or type that do not involve contractions such as absence seizures or complex partial seizures.

EPILEPSY: A group of syndrome characterized by paroxysmal transient disturbances

of brain function its causes seizure or fits.

Status epilepticus accounts for 1-8% of all hospital admissions for epilepsy.

Physiological A seizure involves abnormal electrical activity in the brain affecting both the mind and the body. This is a medical emergency that may lead to

(6)
(7)

Pathophysiology

Status epilepticus-common etiology

Status epilepticus may occur in those with a history of

epilepsy only

25%

as well as those with an underlying problem of the

brain

. These

underlying problems include

Trauma

Infections

Stroke, including hemorrhagic

Alcohol withdrawal

Anoxic brain injury

Metabolic disturbances (such as affected liver and kidney)

Metabolic derangements(shock)

o

Hypoxia, Hypoglycemia ,Hyponetremia, hypomagnesaemia

(8)

Conti…

 Head Injuries  Meningitis  Deliriumme

Intoxication or adverse reactions to

Drugs

o

Antibiotics ( pencilline's ,isoniazid)

o

Anasthetics, norcotics (halothane ,cocain, ketamine)

o

Psychopharmaceuticals (antidepresent , antipsychotics , antidepresent

Insufficient dosage or sudden withdrawal of a medication (especially anticonvulsants)Dieting or fasting while on an anticonvulsant

A new medication that reduces the effectiveness of the anticonvulsant its half life

leading to decrease blood concentration.

(9)

Pathogenesis

Message from body carried by neuron

Control discharge or electrochemical energy

Impulses occur in bursts whenever a cell has a task to perform

If EC discharge is un control or abnormal

A person said to having an epileptic syndrome

If prolong epileptic seizures

(10)
(11)

Pathogenesis

Many neurons fire in a synchronous pattern, resulting in a transient

physiologic disturbance .

The risk of cell injury depends also on the overall pathophysiologic profile,

including the presence of alterations resulting from SE and occurring independent of SE.

On neurophysiologic grounds. we divide SE into "spike-wave" and

"nonspike-wave" forms. Spike-wave "absence" status epilepticus carries a low risk of epileptic brain damage, and therapy should be adjusted accordingly. All

nonspike-wave SE has a theoretical basis for epileptic brain damage, but the actual risk is variable.

There is a significant known risk of cell injury during generalized convulsive SE,

(12)

Clinical Manifestation (Signs and symptoms)

Non-Convulsive SE

convulsive SE

IN convulsive status epilepticus

Unusual behavior leads to

Confusion and stiff body with

eyes rolled upward and dilated

pupils and Muscle spasms is

major sign.

Difficulty speaking

Falling

Unusual noises

Loss of bowel or bladder

control

Clenched teeth

Irregular breathing

A "daydreaming" look

This is an extended seizure with no

physical convulsions. Symptoms of status

epilepticus without outward physical signs

are unresponsiveness, confusion or

agitation, and confirmed by EEG

(13)

Convulsive SE

Increased CO

Increased BP

Increased BS

Increased Lactate levels

Decomposition-Failure of Homeostatic failure

Reduced CO- Levels/BS/Lactate/Oxygen levels leads

to:

1.

Cardiorespiratory collapse

2.

Electrolyte Imbalance

3.

Hyperthermia

(14)

Diagnostic criteria

Routine labs( lactic acidosis)

ABG’s(acidosis)

CT Scan brain

MRI

EEG

(15)

First Aid for Seizures:

If you see someone having a seizure, take the following

steps

Time the seizure with your watch.

Clear the area of anything hard or sharp.

Loosen anything at the neck that may impair breathing.

Turn the person onto his or her side.

Put something soft beneath the head.

Do not place anything inside the mouth.

Call Area Emergency Number, if a seizure lasts more than

(16)

MEDICAL MANAGEMENT

Emergency/inpatient management includes basic life support (0-10 minutes) and

pharmacological management (10-60 minutes).

Airway & Oxygenation is established (pass ETT if unconscious , if need ventilate)

Anti epileptic medication(keep seizure free)

Anti sedative drugs

IV Fluid ( 5% dextrose if hypoglycemic)

Thiamaine 100mg proior to dextrose

Monitor frequently S\E & Glucose

Continue monitor neurologic sign

Drugs used in pharmacological management are Injectibale lorazepam, midazolam,

(17)

Surgical management

Common surgical procedure for treatment of seizure is cortical exicision i.e lobectomy

When temporal lobe epilepsy, then resection of the antero-medial temporal lobe called

mesial temporal lobectomy

If scar tissue or other focal epileptogenic area exists the identified lesion

(lesionectomy) can be removed

A corpus callosotomy has been helpful in patients with tonic clonic seizures

A hemispherectomy is reserved for selected catastrophic infant and early childhood

epilepsies

Other therapies

Vagal nerve stimulation

An electrode is surgically placed around the left vagus nerve in the neck. It is

(18)

Nursing Diagnosis

Nursing Diagnosis

Risk for Trauma

Risk for Suffocation

Risk factors may include

Weakness, balancing difficulties; reduced muscle, hand or eye coordination

Poor vision

Reduced sensation

Cognitive limitations or altered consciousness

Loss of large or small muscle coordination

Emotional difficulties

Possibly evidenced by

Not applicable. A risk diagnosis is not evidenced by signs and symptoms, as the problem has

(19)

Nursing Management

The nursing goal is to prevent injury to the patient. This includes not only

physical support but psychological support as well.

Provide privacy, Ease the patient on the floor, if possible to protect the head

with a pad to prevent injury, Place the patient on one side with head flexed

forward. Loosen constrictive clothing.

If aura precedes the seizure, place padded tongue blade between the teeth

Do not attempt to pry open jaws that are clenched in a spasm to insert

anything

Clear airway

Oxygen therapy

Telemetry

Iv access

Instruct about medications such as anti epileptic

(20)

Classification

Systemic

Complications

Cardiac

Hypertension, tachycardia (reversing after 30 minutes),arrhythmias,

cardiac arrest

Pulmonary

Apnea, respiratory failure, hypoxia, neurogenic pulmonary edema,

aspiration pneumonia

Autonomic

Fever, sweating, hyper secretion (including tracheobronchial),vomiting

Metabolic

Hyperkalemia, hyperglycemia then hypoglycemia, volume depletion,

venous stasis, possible thrombosis

(21)

Cerebral

Neuronal damage similar to that of hypoxia, hyperthermia : cortical layers 3 and

5, cerebellum, and hippocampus

Cerebral edema, raised intracranial pressure

Cortical vein thrombosis

Neurologic squeals

I

ncreased seizure

frequency

, recurrent status epilepticus

Decreased cognitive function (controversial)

(22)
(23)

ANATOMICAL OVERVIEW OF RESPIRATORY SYSTEM

Upper Respiratory Tract Lower Respiratory Tract

Nose

Para nasal sinuses

Pharynx

Tonsils

Larynx

Trachea

Lungs

o

Pleura

o

Media sternum

o

Bronchi &

bronchioles

(24)
(25)

FUNCTION OF RESPIRATORY SYSTEM

Oxygen transport

Respiration

Ventilation

Pulmonary perfusion & diffusion

(26)

Status Asthamaticus

Asthama

A respiratory

condition marked by

attacks of spasm in

the bronchi of the

lungs , causing

difficulty in breathing

are often due to

allergic reaction or

others forms of

hypersensitivity.

Status Asthamaticus

A prolonged sever attack of

asthma that is unresponsive to

initial stander therapy, is

characterized especially by

dispend, dry cough , wheezing &

hypoxemia that may lead to

(27)
(28)

PATHOPHYSIOLOGY

Irritant or terrors Inflamed bronchial mucosa Decrease diameter of the bronchi

Decrease PaO2 & PCO2

(29)
(30)

SIGN & SYMPTOMS OF STATUS

ASTHMATICUS

Labored

breathing &

wheezing

Prolonged

exhalation

Respiratory

failure

Obstruction

worsens

Engorged

(31)
(32)

Causes of status Asthamaticus

Air pollution

Allergens

climate

Respiratory

Infection

Physical and

(33)

ASSESSMENT & DIAGNOSIS

Respiratory assessment

Routine labs ( CBC basophils increase)

(34)

MEDICAL MANAGMENT

Short acting beta2 aderenegenic agonist (rapid relief

bronchospasm)

Corticosteroids(

reduce inflammation)

Oxygen therapy(to treat cyanosis, dyspnea & hypoxemia)

IV fluid and hydration

Sedative are contraindicated

Muscle relaxation (magnesium sulphate & a calcium

antagonist)

Treat respiratory acidosis ( use Bipap , may need mechanical

(35)

NURSING DIAGNOSIS

Impaired gas exchange

related to ventilation perfusion inadiquality

Ineffective airway clearance

related to increase mucous production

or ineffective cough

Ineffective breathing pattern

related to shortness of breath

Self care deficits

related to fatigue secondary to increase work of

breathing

(36)

NURSING MANAGMENT

Asses the airway & patient response to treatment

Telemetry until the sever exercitation resolve

IV access (3 to 4L\ day

Patients room should be quite & free from

irritant.

(37)

COMPLICATIONS

Pneumonia

Atelectasis

Sever dehydration

Airway obstruction

(38)

References

Text book of medical surgical nursing ( Brunner & suddarth,s)

www.medscape.com

http://emedicine.medscape.com/article/2129484-treatment

http://

www.epilepsy.com/information/professionals/resource-library/tables/compli

cations-status-epilepticus

http://www.hopkinsmedicine.org/healthlibrary/conditions/nervous_system_d

(39)

ANY

QUE

(40)

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