HPV 16/18
Determinants of seropositivity among HPV 16/18 DNA positive young women
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Cervarix™: a vaccine for the prevention of HPV 16, 18-associated cervical cancer
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Introduction and sustained high coverage of the HPV bivalent vaccine leads to a reduction in prevalence of HPV 16/18 and closely related HPV types
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Impfung gegen humane Papillomaviren (HPV) zur Prvention HPV 16/18 induzierter Zervixkarzinome und derer Vorstufen Human papillomavirus (HPV) vaccination for the prevention of HPV 16/18 induced cervical cancer and its precursors
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Comparison of Human Papillomavirus Detection by Aptima HPV and cobas HPV Tests in a Population of Women Referred for Colposcopy following Detection of Atypical Squamous Cells of Undetermined Significance by Pap Cytology
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Original Article Tenascin-C is a diagnostic marker for cervical cancer and a potential marker for cancer-associated fibroblasts in cervical carcinoma
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Prevalence of HPV After Introduction of the Vaccination Program in the United States
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Potential Natural Inhibitor Bioactivities against High-Risk HPV-16, HPV- 18 and HPV-52 of E6 Oncoprotein through Molecular Docking Vivitri D. Prasasty*, Ignatia Eveline, Ekaristy J. Noya
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Human papillomavirus (HPV) types 16, 18, 31, 45 DNA loads and HPV 16 integration in persistent and transient infections in young women
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A sensitive, type specific, fluorogenic probe assay for detection of human papillomavirus DNA
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HPV vaccination programs have not been shown to be cost-effective in countries with comprehensive Pap screening and surgery
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Co occurrence of human papillomavirus (HPV) in newborns and their parents
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Progression and regression of incident cervical HPV 6, 11, 16 and 18 infections in young women
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Results of the First World Health Organization International Collaborative Study of Detection of Human Papillomavirus DNA
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Human Papillomavirus Virus-Like Particles Are Efficient Oral Immunogens when Coadministered with Escherichia coli Heat-Labile Enterotoxin Mutant R192G or CpG DNA
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The viral transcriptional regulatory region upstream of the E6 and E7 genes is a major determinant of the differential immortalization activities of human papillomavirus types 16 and 18.
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A viral-cellular junction fragment from a human papillomavirus type 16-positive tumor is competent in transformation of NIH 3T3 cells.
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Enhanced transcriptional activation by E2 proteins from the oncogenic human papillomaviruses.
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Human Papilloma Virus (HPV) in squamous cell carcinoma of the oesophagus
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